type I cells - traduction vers arabe
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type I cells - traduction vers arabe

WIKIMEDIA DISAMBIGUATION PAGE
I cells; I-cells; I cell
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type I cells      
‎ الخَلاَيا السِّنْخِيَّةُ مِنَ النَّمَطِ الأَوَّل‎
tyrosinosis         
  • Tyrosinemia type I has an autosomal recessive pattern of inheritance
  • Pathophysiology of metabolic disorders of tyrosine, resulting in elevated levels of tyrosine in blood.
  • Nitisinone
  • Tyrosine metabolic pathway. Fumarylacetoacetate hydrolase (FAH) is shown to be nonfunctional, leading to the accumulation of maleylacetoacetate (MAA) and succinylacetoacetate (SAA), the later of which is converted to succinylacetone (SA).
  • Tyrosine
TYROSINEMIA THAT HAS MATERIAL BASIS IN DEFICIENCY OF THE ENZYME FUMARYLACETOACETATE HYDROLASE RESULTING IN AN INCREASE IN FUMARYLACETOACETATE WHICH INHIBITS PREVIOUS STEPS IN TYROSINE DEGRADATION LEADING TO AN ACCUMULATION OF TYROSINE IN THE BODY
Type I tyrosinemia; Hepatorenal tyrosinemia; Tyrosinemia type 1; Tyrosinosis; Draft:Tyrosinemia (Type I); Tyrosinemia (Type I)
الدَّاءُ التِّيروزينيّ
tyrosinosis         
  • Tyrosinemia type I has an autosomal recessive pattern of inheritance
  • Pathophysiology of metabolic disorders of tyrosine, resulting in elevated levels of tyrosine in blood.
  • Nitisinone
  • Tyrosine metabolic pathway. Fumarylacetoacetate hydrolase (FAH) is shown to be nonfunctional, leading to the accumulation of maleylacetoacetate (MAA) and succinylacetoacetate (SAA), the later of which is converted to succinylacetone (SA).
  • Tyrosine
TYROSINEMIA THAT HAS MATERIAL BASIS IN DEFICIENCY OF THE ENZYME FUMARYLACETOACETATE HYDROLASE RESULTING IN AN INCREASE IN FUMARYLACETOACETATE WHICH INHIBITS PREVIOUS STEPS IN TYROSINE DEGRADATION LEADING TO AN ACCUMULATION OF TYROSINE IN THE BODY
Type I tyrosinemia; Hepatorenal tyrosinemia; Tyrosinemia type 1; Tyrosinosis; Draft:Tyrosinemia (Type I); Tyrosinemia (Type I)
‎ الدَّاءُ التِّيروزينيّ‎

Définition

type specimen
¦ noun Botany & Zoology the specimen, or each of a set of specimens, on which the description and name of a new species is based. See also holotype, syntype.

Wikipédia

I-cell

Fritz Heinrich Jakob Lewy, a German-American neurologist, first identified and described inclusions in the brain cells of patients with Parkinson’s disease and published his findings in the Lewandowsky’s Handbook of Neurology in 1912. I-cells also called inclusion cells are abnormal fibroblasts having a large number of dark inclusions in the cytoplasm of the cell (mainly in the central area). They are metabolically inactive structures of a cell and are not enclosed by a membrane. The inclusions are of various fats, proteins, carbohydrates, pigments, excretory products, crystals, and other insolubles. They are found in the cytoplasm of a cell in both prokaryotes and eukaryotes. They are seen in Mucolipidosis II, and Mucolipidosis III, also called inclusion-cell or I-cell disease where lysosomal enzyme transport and storage is affected.